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Providing independent clinical excellence since 2005

The SCVC Blog

Cardiovascular care news and articles from our expert team

Statins, Absolute Benefit, and the Problem of Residual Risk

When people think about cholesterol, they often imagine it as something floating in the blood, disconnected from body fat. In reality, the type and location of body fat — particularly visceral fat (VAT) — profoundly influence how the liver packages and clears cholesterol. Raised VAT doesn’t just sit silently around the organs; it acts like an endocrine organ, sending chemical distress signals that shift lipid metabolism in an atherogenic direction.

Exercise and Digital Tools Should Be the First Line in Reducing Visceral Fat in Cardiac Patients

We use waist-to-height ratio (WHtR) as a screening tool, followed by low-dose CT to measure VAT index (VATI) against gender- and ethnicity-specific thresholds. Where VATI is high, patients enter a six-month digital lifestyle programme using tools like the Dr Shape food app, CGM, and body composition scales. Pharmacological therapy, including GLP-1 receptor agonists, is reserved for non-responders—making this a precise, cost-effective, and highly personalised pathway for VAT reduction.

Why Cardiovascular Prevention Matters Before 30

Emerging evidence shows that lifetime exposure to LDL cholesterol and high glucose variability are some of the strongest predictors of cardiovascular risk. Just as lung cancer risk is tied to pack-years of smoking, heart disease risk correlates with cumulative LDL levels and glucose metrics such as HbA1c over time.This is why some people in their 20s with risk factors (like diabetes or a family history of heart disease) are starting statin therapy preventatively. However, many are understandably hesitant to start medication so early. Lifestyle changes remain the cornerstone of early prevention.

How a daily junior aspirin can save millions of lives

Aspirin, or acetylsalicylic acid, is one of the oldest and most widely used medications in the world. Well known for its analgesic effects, it was later discovered to be effective in much smaller doses in preventing platelet aggregation that had a pivotal role in arterial thrombosis. Its  role in inhibiting platelet activity has made it a cornerstone in the prevention and treatment of arterial thrombosis. In this article, we will explore the fascinating role of platelets in acute coronary thrombosis, the mechanisms by which aspirin exerts its protective effects, and the clinical considerations for its use.