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An article written by Dr Edward Leatham, Consultant Cardiologist Tags: BP, CGM, Glucose, Telehealth, Homecare, NH1 search website using Tags to find related stories. Over the past decade, healthcare has experienced a remarkable evolution in how patients are monitored and managed […]
Having a stent or bypass is not the end of the story—it is the beginning of a new chapter in managing your cardiovascular health. With the right combination of medications, lifestyle changes, rehabilitation programmes, and vigilant follow-up using advanced diagnostic techniques, you can greatly reduce your risk of future events. Stay engaged with your healthcare team, keep informed about new therapies and guidelines, and commit to sustainable lifestyle adjustments to ensure the best possible long-term health.
To understand how beta-blockers achieve their effects, imagine the heart as a van engine with a speed regulator fitted and adrenaline as the driver pressing down hard on the accelerator pedal. When you take a beta-blocker, it prevents the engine from racing even when the driver presses the accelerator hard down. The result is a slower top speed and, steadier “drive”—your heart beats more slowly, and the strain on the engine (your heart muscle) reduces.
A key component of evaluating statin-related symptoms is tracking them systematically. A symptom diary can help you visualise the severity and frequency of side effects over time. A "year-at-a-glance" dot plot is particularly useful.
Aspirin, or acetylsalicylic acid, is one of the oldest and most widely used medications in the world. Well known for its analgesic effects, it was later discovered to be effective in much smaller doses in preventing platelet aggregation that had a pivotal role in arterial thrombosis. Its role in inhibiting platelet activity has made it a cornerstone in the prevention and treatment of arterial thrombosis. In this article, we will explore the fascinating role of platelets in acute coronary thrombosis, the mechanisms by which aspirin exerts its protective effects, and the clinical considerations for its use.
The timing of intervention in aortic stenosis is critical. Delaying surgery or transcatheter procedures until symptoms become severe or complications arise can lead to poorer outcomes. Conversely, intervening too early may expose patients to unnecessary procedural risks. For exceptionally fit individuals aged 75-85, timely intervention can be particularly advantageous, as they may tolerate surgical aortic valve replacement (SAVR) better than expected for their age. In these cases, earlier treatment may prevent irreversible damage to the heart and reduce the risks associated with waiting until the disease progresses further.
Aortic stenosis (AS) is a progressive and potentially life-threatening condition. It predominantly affects older adults and has significant implications for cardiovascular health, life expectancy, and quality of life. The incidence of AS is climbing because of increased life expectancy. Timely intervention, especially in symptomatic patients or those with progressing stenosis, is essential to optimise outcomes. This article explores the nuances of AS diagnosis, treatment decisions, and the importance of patient involvement in the care pathway.
Angina is not a disease in itself but a symptom of underlying cardiac problems. It is important for both patients and healthcare providers to recognise that angina acts as a warning sign of the heart’s distress. Typically manifesting during activities that increase the heart’s demand for oxygen, such as physical exertion or emotional stress, the anginal symptom indicates that the heart is not receiving enough oxygenated blood.
An article written by Dr Edward Leatham, Consultant Cardiologist Tags: Podcast, AFib, Bradycardia, NH1 search website using Tags to find related stories. Introduction Pulse rate or heart rate is a fundamental metric of heart health. It’s easily accessible—whether through feeling the […]
This post is not for the faint hearted! It is a technical description of how the heart works, and may help some readers/listeners to understand the difference between HFpEF and HFrEF. The main objective of this blog is to emphasise the enormous potential of reducing left ventricular stiffening as we age. This can only be accomplished by paying attention to key health metrics from mid life, all influenced by lifestyle, diet and physical exercise
A slow heart rate may not cause symptoms in many people. However, when bradycardia leads to insufficient blood flow, symptoms may arise. The causes of bradycardia are discussed and what constitutes a normal range. An associated podcast is available too.
In atrial fibrillation or AFib, the atrial tissue quivers at approximately 600 cycles per minute, which means that it has no useful pumping function. The left atrium therefore, in patients with atrial fibrillation, merely acts as a passive conduit between the lungs and the left ventricle. It no longer serves to prime the left ventricle, and as a result, less blood enters the heart during diastole, less 'priming' occurs so cardiac output may fall. If ever you have experienced a car's turbo charger going 'on the blink' you will know that the car still GOES, but not very well, since it too suffers loss of output. The left atrial pressure in many such cases will increase, causing oedema or fluid in the alveolar space, reducing gas exchange and contributing to the symptom of breathlessness.